Current research projects include human and animal investigations into individual differences in the biobehavioral effects of stress.
Our human studies focus on the examination of
sex
differences in neuroendocrine and immunologic responses to stress in humans.
In
particular, we are interested in the role of posterior pituitary hormones, such
as oxytocin, and cytokines (IL-1B and IL-6) in moderating biobehavioral responses to
stress in men and women. This research, funded by the National
Science Foundation, is based on a new stress theory -- a biobehavioral
response we label “tend-and-befriend” (Taylor, Klein, et al., 2000), which
is a biobehavioral pattern of stress responses that includes the creation and
maintenance of social networks and nurturant activities designed to protect the
individual and their offspring.
Our animal research is designed to investigate biological stress mechanisms that are difficult to study in humans. Specifically, we design animal models that are believed to parallel the human stress condition. We currently are investigating the influence of adolescent nicotine exposure (a potent biological stressor) on drug abuse in adulthood. Our laboratory was the first to develop an animal model of the epidemiologic report that kids who smoke are more likely to use drugs as they get older (Klein, 2001). Together with Dr. David Vandenbergh from the Department of Biobehavioral Health and the Center for Developmental and Health Genetics, we now are examining gene responses to nicotine exposure in reward-relevant brain regions to determine underlying mechanisms for the development of drug addiction.